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Welcome, Clinicians!

At Cindnesskult.com, our mission is to foster a deeper understanding of mental health through an integrative, developmental lens. This resource page is designed to support you in providing comprehensive, nuanced, and effective care by offering practical guidelines and theoretical insights.

Why This Resource?

In the complex landscape of mental health, accurate assessment and tailored intervention are paramount. This guide consolidates essential protocols for initial client sessions, introduces "The Interwoven Roots Theory of Psychopathology" as a framework for understanding symptom manifestation, and provides practical tools for differential diagnosis. Our aim is to streamline your process, enhance diagnostic precision, and ultimately contribute to better client outcomes.

Helpful Tips for Using This Guide

From the Interwoven Roots Theory: The Developmental Model of Psychopathology
Vivian Mitchell, MA, APCC 16067, Ph.D. Student of Clinical Psychology
(Preprint Finalization)

Developmental Pathways & Symptom Manifestation

This model outlines the developmental progression from foundational roots to symptomatic clusters. The pathways are not rigid but represent common patterns of how vulnerabilities manifest.

Level 3: Full Symptom Development in Poor Circumstances/Complex Presentations
Primarily AE-Driven:
Dissociative Identity Disorder (DID), Borderline Personality Disorder (BPD), Complex PTSD
Primarily Neurodev/Genetic:
Schizophrenia Spectrum, Bipolar I & II (severe)
Primarily Interactional:
Severe Generalized Anxiety Disorder (GAD), Major Depressive Disorder (MDD), Obsessive-Compulsive Disorder (OCD); Personality Disorders
Level 2: More Specified, Complex, or Progressive Disorders
Primarily AE-Driven:
Treatment-resistant MDD/Anxiety, severe/enduring Eating Disorders, DID/OSDD, deeply entrenched Personality Disorders
Primarily Neurodev/Genetic:
Schizophrenia, Bipolar I (severe)
Primarily Interactional:
Severe GAD, MDD, OCD; Personality Disorders (often with multiple interacting roots, cumulative AEs)
Level 1: Early Diagnosable Conditions / Core Symptom Clusters
From AE:
Adjustment Disorders, PTSD, Trauma-Related OCD, Dissociative Disorders, Borderline Personality Disorder traits, Reactive Anxiety/Depression.
From ADHD:
ADHD, co-occurring Anxiety, Depressive symptoms, early Substance Use Disorders (SUDs) (coping), some Impulse Control issues. AEs can exacerbate.
From ASD:
Avoidant/Restrictive Food Intake Disorder (ARFID), Pica, Social Anxiety, Depression (often due to environmental mismatch/trauma pipeline). Potential overlap with Avoidant Personality Disorder (AVPD), Dependent Personality Disorder (DPD) traits.
From Genes (Other):
Intellectual Disability (ID), Learning Disorders, Primary Sleep-Wake Disorders, Cyclothymic Disorder, Primary OCD, early signs of Bipolar spectrum, Attenuated Psychosis Syndrome (if strong genetic loading).
Level 0.5: Prodromal / Sub-Clinical Manifestations / Early Signs
  • From AE: Early, less differentiated signs of distress or characteristic difficulties emerging from Level 0 Roots. Acute stress, emotional dysregulation, somatic complaints, mood changes, social withdrawal, sleep/appetit disturbances.
  • From ADHD: Mild executive dysfunction, restlessness, emotional over-reactivity, mild cognitive/executive issues.
  • From ASD: Mild sensory sensitivities, social awkwardness, anxiety, heightened sensitivities, early repetitive behaviors.
  • From Genes (Other): Mild temperamental expressions, sub-threshold mood fluctuations.
Epigenetics (The Bridge): The mechanism by which Adverse Events influence gene expression.
Level 0: Foundational Roots / Primary Etiological Domains
Adverse Events (AE):
Trauma, neglect, chronic stress, discrimination, poverty, nutritional deficiencies.
ADHD Neurotype:
Highly heritable neurodevelopmental profile (Inattention, impulsivity, hyperactivity differences).
ASD Neurotype:
Highly heritable profile (Social communication, sensory processing, RRBI differences). Higher risk for AEs (Hoover & Kaufman, 2018).
Genetic/Bio (Other):
Polygenic risks (psychosis, bipolar), MTHFR, neurochemical variations, predispositions for schizophrenia, bipolar, etc.

Treatment Implications & Future Directions

Clinical Vignette: The Case of "Alex"

"Alex" (13) with family history (Genetic predispositions), suspected neurodivergence (ASD/ADHD neurotype), and a history of home abuse (Adverse Events), first presented with bipolar-like symptoms (which were a form of coping). After medication stabilized mood, unaddressed trauma led to new OCD, BPD traits, and finally dissociation (Other Specified Dissociative Disorder - OSDD-1), which became a "safer" coping mechanism than mania. This case illustrates that without addressing the foundational root of safety, the system may generate new, complex symptoms to survive.

Phased & Individualized Treatment Approach

Critique: Modalities like EMDR or CBT can be ineffective or even harmful if applied without a full understanding of the underlying roots and the client's current state.

  1. Safety, Stabilization, & Root Identification: Establish safety, provide psychoeducation, and assess the four foundational roots.
  2. Symptom Tracing & Prevention: Map developmental pathways to understand symptom emergence. This allows for proactive work to prevent further progression by identifying prodromal symptoms (Level 0.5) of other potential clusters and building targeted protective factors.
  3. Addressing Specific Symptomatic Clusters: Once stabilized, use evidence-based interventions to treat the most pressing Level 1, 2 or 3 symptom clusters.
  4. Deeper Trauma Integration: Specialized, trauma-focused work should be initiated after a foundation of safety and stability is established.

Protective Factors & Resilience

These factors can buffer against risk and promote positive adaptation.

Pillar 1: Innate Temperament & Early Regulatory Capacities
Pillar 2: Sense of Community (Ecological Model)
Pillar 3: Attachment Security (and Earned Security)
Pillar 4: Nature of Dissociative Response & Trauma Integration
Pillar 5: Holistic Well-being Practices (Nutrition, sleep, etc.)
Pillar 6: Positive Childhood Experiences (PCEs)
Pillar 7: Developed Psychological Capacities & Skills
Pillar 8: Active Coping & External Engagement (Purpose)

Clinical Application: Proposed Methodological Approach

A. Initial Etiological Exploration

Before assessing specific symptom clusters, it is crucial to explore foundational etiological factors. This involves a multi-faceted approach to understand the client's history and biological context.

  1. Gather Comprehensive History: Systematically collect detailed family history (e.g., using structured interview forms) and medical history to identify genetic predispositions and existing conditions.
  2. Establish Timeline of Onset: Create a clear timeline of when symptoms appeared to rule out causes related to medication changes (iatrogenic), injury, illness, or substance use.
  3. Collaborate with Medical Providers: To rule out biological contributors, request lab testing for vitamin/mineral deficiencies (e.g., Vitamin D, B12) and hormonal imbalances. If possible, consider pharmacogenomic testing (e.g., GeneSight).

"If This, Then That" - Symptom Clusters & Root Exploration

This section explores common symptom clusters and their potential underlying roots, guiding a more targeted and effective clinical approach.

Clinical Caveat on "Forced Coping": A core tenet is addressing foundational roots, particularly active Adverse Events (AEs), before or concurrently with interventions for specific symptomatic clusters. Without addressing the underlying roots, the system may generate new, complex symptoms as a means of survival.

Neurodevelopmental Disorders

Level 0 Roots: ASD Neurotype, ADHD Neurotype, Genes (Other), AEs (prenatal exposure, birth trauma, early neglect).

Level 1 Diagnoses: Autism Spectrum Disorder, ADHD, Intellectual Disability, Specific Learning Disorder, Communication Disorders, Motor Disorders.

Clinical Progression & Considerations: The severity and presentation of these Level 1 conditions are heavily influenced by the presence and severity of AEs. For example, an individual with an ASD neurotype who experiences significant early trauma and attachment disruption is more likely to meet criteria for ASD Level 2 or 3, requiring more substantial support, compared to an individual with the same neurotype but with strong protective factors.

If a Neurodevelopmental Disorder is present/suspected: Screen for AEs (high risk), Anxiety, Depression, OCD, Eating Disorders (ARFID), and features of Cluster A/C personality disorders as potential maladaptive coping strategies.

Schizophrenia Spectrum and Other Psychotic Disorders

Level 0 Roots: Primarily Genes (Other), with AEs (especially trauma) and nutritional deficiencies acting as significant triggers or accelerators.

Level 1 Diagnoses: Brief Psychotic Disorder, Attenuated Psychosis Syndrome.

Level 2/3 Progression: Untreated Level 1 conditions, especially in the context of ongoing AEs or lack of protective factors, can progress to Schizophreniform Disorder, and then to Schizophrenia or Schizoaffective Disorder (if mood episodes are prominent).

If Psychosis is present: Investigate family history of psychosis/bipolar. Screen thoroughly for trauma (AEs). Rule out Substance-Induced Psychosis. Carefully assess for underlying ASD, as sensory experiences or communication differences can be misdiagnosed as psychosis.

Bipolar and Related Disorders

Level 0 Roots: Primarily Genes (Other). AEs are a major trigger, often influencing an earlier age of onset and more severe course.

Level 1 Diagnoses: Cyclothymic Disorder.

Level 2/3 Progression: Cyclothymia, particularly when compounded by AEs, can progress to Bipolar II, and subsequently to Bipolar I. The presence of significant trauma can lead to presentations with rapid cycling, mixed features, and psychotic features, sometimes evolving into a Schizoaffective Disorder presentation.

If Bipolar symptoms are present: Screen for AEs/trauma. Assess for co-occurring ADHD, Anxiety Disorders, and SUDs.

Depressive Disorders

Level 0 Roots: Can stem from any of the four roots (AEs, Neurodivergence, Genes).

Level 1 Diagnoses: Persistent Depressive Disorder (Dysthymia), Unspecified Depressive Disorder.

Level 2/3 Progression: Chronic, untreated depression, especially when rooted in trauma or neurodivergence, can become Major Depressive Disorder, single or recurrent episode, with increasing severity specifiers.

If Depression is present: Screen for AEs, ADHD, ASD (burnout/social isolation), and rule out medical/nutritional causes. Crucially, screen for a history of mania/hypomania to rule out Bipolar Disorder.

Anxiety Disorders

Level 0 Roots: Can stem from any of the four roots.

Level 1 Diagnoses: Unspecified Anxiety Disorder, Generalized Anxiety Disorder.

Level 2/3 Progression: Can progress to more specific and impairing forms like Panic Disorder, Agoraphobia, and Social Anxiety Disorder. Social Anxiety in the context of ASD may be a reasonable response to a lifetime of social difficulties, not a disordered perception.

If Anxiety is present: Screen for AEs, ADHD, ASD. Differentiate GAD from the worry/anxiety inherent to OCD.

Obsessive-Compulsive and Related Disorders

Level 0 Roots: Genes (Other), AEs (trauma-related OCD), ADHD/ASD (as a coping mechanism).

Level 1 Diagnoses: OCD.

Level 2/3 Progression: Untreated OCD, especially when co-occurring with ADHD or significant trauma, can increase in severity, leading to specifiers like "with poor insight" or "with absent insight/delusional beliefs."

If OCD is present: Assess for underlying ADHD/ASD. Explore trauma history. Differentiate from OCPD traits.

Trauma- and Stressor-Related Disorders

Level 0 Roots: Primarily AEs. Neurodivergence can increase vulnerability to trauma.

Level 1 Diagnoses: Adjustment Disorder, Acute Stress Disorder, PTSD.

Level 2/3 Progression: Chronic or complex PTSD is a Level 2/3 condition. If PTSD is present, it is critical to screen for the development of Dissociative Disorders (OSDD/DID) and Personality Disorders, particularly BPD.

If PTSD is present: Screen for Dissociative Disorders, BPD, SUDs, and underlying neurodivergence.

Dissociative Disorders

Level 0 Roots: Primarily severe, chronic childhood AEs (trauma).

Level 1/2 Diagnoses: Depersonalization/Derealization Disorder, Other Specified Dissociative Disorder.

Level 3 Progression: These are often Level 3 diagnoses by nature, particularly Dissociative Identity Disorder, representing a profound adaptation to overwhelming trauma.

If Dissociation is present: A thorough trauma history is essential. Assess for co-occurring PTSD, BPD, Depression, and Anxiety.

Personality Disorders

Level 0 Roots: Primarily AEs interacting with Genes (Other)/Neurotype.

If PD traits are present: Conduct a thorough trauma history. Assess for underlying ASD/ADHD. The presence of BPD strongly suggests a history of trauma and attachment disruption. Traits of AVPD and DPD often overlap significantly with the presentation of an unaccommodated autistic individual. Low empathy temperamental traits interacting with AEs are a key pathway to NPD and ASPD.

Key Diagnostic Considerations & Screening Tools

This section provides guidance on essential screening tools and crucial differential diagnostic considerations for various clinical presentations.

Suggested Screening Tools for Symptom Clusters

The following tools can be used to screen for specific symptom clusters. Most can be self-administered, but some require clinical training for interpretation.

Domain Tool(s) Est. Time Notes Location
General Distress DASS-10, PHQ-9, GAD-7 ~5 mins DASS for initial screen. PHQ-9/GAD-7 for ongoing tracking/confirmation. Client Portal, EHR
Trauma / PTSD ACE-Q, ITQ, PCL-5 5-15 mins ACEs for history. ITQ for C-PTSD. PCL-5 for PTSD. Client Portal
Autism AQ-10 ~5 mins Pre-screen. Requires clinical training for interpretation. Client Portal
ADHD ESQ-R, Vanderbilt, Conners 5-30 mins Vanderbilt/Conners for children (short form ~5 min, full ~20-30 min). ESQ-R for adolescents/adults. Client Portal
Bipolar Disorder MDQ ~5 mins Rule out within 5 sessions. Requires clinician interpretation. Client Portal
Psychosis PQ-B, PANSS 5-10 mins PQ-B for screening. PANSS for full assessment (requires specialized training). Internal Resources
OCD OCI-R, ChOCI-R (Child) ~10 mins Rule out within 5 sessions. High "ordering" may suggest neurodivergence. Client Portal
Dissociation MID ~20 mins Multidimensional Inventory of Dissociation. Client Portal
Personality PID-5-SF ~15 mins Personality Inventory for DSM-5, Short Form. Client Portal
Anxiety (Child) RCADS, SCARED 10-15 mins Screens for various anxiety and depressive disorders in youth. Client Portal
Substance Use IRIS, AUDIT, DAST 5-10 mins Initial screen for alcohol and drug use risk. Client Portal

Key Diagnostic Rule-Outs & Exclusions (DSM-5-TR Informed)

Bipolar vs. Depressive Disorders: A history of a manic, hypomanic, or mixed episode excludes a diagnosis of Major Depressive Disorder, Persistent Depressive Disorder (Dysthymia), or other primary depressive disorders. The presence of mania or hypomania indicates a fundamental difference in mood regulation that is distinct from unipolar depression.

Schizophrenia Spectrum vs. Mood Disorders: These primary psychotic disorders generally exclude a diagnosis of a Depressive Disorder or Bipolar II Disorder/Cyclothymic Disorder if the mood episodes are brief relative to the total duration of the psychotic or delusional periods. Schizoaffective Disorder is the exception, as it requires a mood episode concurrent with psychotic symptoms, along with a period of psychosis without a major mood episode.

Substance/Medication-Induced vs. Primary Disorder: If the symptoms of a mental disorder are judged to be entirely and directly caused by a substance, medication, or medical condition, a primary mental disorder diagnosis cannot be given. Instead, the appropriate Substance/Medication-Induced or Due to Another Medical Condition diagnosis is used. Symptoms occurring exclusively during intoxication or withdrawal are classified as Substance/Medication-Induced and would exclude a primary diagnosis if the substance use is the sole cause.

Autism Spectrum Disorder (ASD) vs. Personality Disorders: If criteria for ASD are met, a diagnosis of Schizotypal Personality Disorder or Schizoid Personality Disorder should not be made unless there is a marked disparity between social communication deficits and other behaviors not typical of ASD. With Narcissistic (NPD) and Antisocial (ASPD) Personality Disorders, clinicians must carefully differentiate if the traits stem from core ASD social cognition differences or a disregard for others. A dual diagnosis requires that criteria for both are distinctly met and not better explained by the other condition.

ADHD vs. Other Mental Disorders: The criteria for ADHD state that symptoms should not occur exclusively during the course of Schizophrenia or another Psychotic Disorder and are not better explained by another mental disorder. However, ADHD can be diagnosed concurrently with conditions like Bipolar Disorder or ASD if criteria for both are fully met and the ADHD symptoms are not solely a manifestation of the other disorder.

OCD vs. Psychotic Disorders: While OCD can have "poor insight" or "delusional intensity" regarding obsessions, the delusions are typically limited to the content of the obsessions. If psychotic symptoms are prominent, persistent, and not exclusively related to the OCD themes, a separate Psychotic Disorder might be considered.

Diagnoses for Careful Consideration/Consultation

The following diagnoses often benefit from consultation with a supervisor or specialist before being finalized:

  • Bipolar I & II Disorders
  • ADHD (all presentations)
  • Obsessive-Compulsive Disorder (OCD)
  • Borderline Personality Disorder (BPD)
  • Schizoaffective Disorder
  • Schizophrenia
  • Autism Spectrum Disorder (ASD)

Note: It is generally recommended to refer out for all Eating Disorders (e.g., Anorexia, Bulimia) to specialized care.

First Session Guide for Clinicians

This guide outlines two models for conducting initial sessions, adapting to whether pre-screening tools are completed prior to the session or need to be administered during the session.

Ideal Intake

(Screeners Pre-Completed)

Total Estimated Time: 50-60 minutes

Welcome & Logistics

5 min
  • Go over: Confidentiality, policies, supervisor info.

Presenting Concern & History

15-20 min
  • "What brings you in today?"
  • "How are these symptoms affecting your life? When did they start?"
  • "Have you received treatment before?"

Review Screeners & Risk

10-15 min
  • Review pre-completed forms (e.g., ACE-Q, PHQ-9).
  • Ask clarifying questions about any flagged items.
  • Complete live risk assessment.

Goals & Strengths

10-15 min
  • "What are you hoping to get out of therapy?"
  • "Who do you have for support? What are you proud of?"

Wrap-Up & Next Steps

5 min
  • Summarize, answer questions, and schedule.

Contingency Model

(In-Session Screeners)

Total Estimated Time: 50-60 minutes

Welcome & Presenting Concern

10 min
  • Go over: Confidentiality, policies, supervisor info.
  • "What brings you in today?" / “How are these symptoms affecting your daily life?”

Required Screenings Checklist

15 min
  • ACE-Q: Trauma history.
  • PHQ-9: Depression symptoms.
  • IRIS/CAGE/AUDIT: Substance use risk.
  • DASS-10/GAD-7: Anxiety and stress.

History & Medical/Substance Use

10 min
  • "Have you ever felt this way before? Or received treatment?"
  • "Any family history of mental health or substance use concerns?"
  • "Any psychiatric medications or significant medical conditions?"

Goals & Strengths

10 min
  • "What are you hoping to get out of therapy?"
  • "If the problem was gone tomorrow, what would be different?"
  • "Who do you have for support? What are you good at or proud of?"

Risk Assessment

5 min
  • "Sometimes when people feel overwhelmed, they have thoughts of hurting themselves. Has that ever been the case for you?"
  • "Have you ever had thoughts of hurting someone else?"

Wrap-Up & Next Steps

5 min
  • Briefly summarize, answer client questions, and schedule next session.

Part 1: Initial Session Protocol

Administrative Tasks

  • Confirm Intake Packet is fully completed and signed.
  • Verbally confirm Address and DOB are accurate in the client's file.
  • Check Electronic Health Record (EHR) for Insurance Card; photograph if needed.

Reviewing Cancellation Policy

  • First late cancellation/no-show is a one-time courtesy.
  • Cancellations with >24 hours' notice are not charged.
  • Subsequent late cancellations are billed the full session fee.

Diagnosis

  • Establish a provisional diagnosis. Refer to the full intake protocol for guidance on diagnostic restrictions (e.g., for single-incident trauma).
  • Also include the ICD-10-CM code Z13.89 (Encounter for screening for other disorder) as part of the initial diagnostic call.

Standard Clinical Screenings

For ALL Clients

  • ACE-Q: Adverse Childhood Experiences (Approx. 5 mins)
  • DASS: Depression, Anxiety, Stress Scales (Approx. 5 mins)
    • DASS-10 for ages 12+
    • DASS-Y for ages under 12

IN ADDITION For Clients Aged 12+

  • PHQ-9: Depressive Symptoms (Approx. 5 mins)
  • IRIS: Substance & Alcohol Use (Approx. 5 mins)

Part 2: Follow-Up Assessments (Within 5 Sessions)

Based on clinical presentation, administer the following as needed to refine diagnosis and treatment.

If Anxiety is a primary concern...

Administer the OCI-R (Obsessive-Compulsive Inventory-Revised) to assess for or rule out OCD. (Approx. 5-10 mins)

If Depression is a primary concern...

Administer the MDQ (Mood Disorder Questionnaire) to rule out bipolar disorder.
Note: This is clinician-administered. Ensure you are up-to-date on protocol. (Approx. 5-10 mins in person)

Important Note on Specialized Assessments

Neurodevelopmental Assessments (ADHD/ASD)

This is typically a lengthy process that begins with an initial assessment with a specialized clinician.

Insurance for clients aged 21 and under will typically cover these developmental assessments. For adult clients, insurance typically does not cover this type of specialized assessment.